Aortic dissection is part of the acute aortic syndrome. Dissection was described for the first time during the autopsy of the British king George II in the 18th C. The disease represents blood entering between the layers of the aortic wall, leading to the formation of two parts of the lumen - a true and a false one. A lot of predisposing factors threaten the aortic wall entirety and are a reason for dissection onset: arterial hypertension, hereditary connective tissue disease (Marfan, Ehlers-Danlos), atherosclerosis, vasculitis, iatrogenic factors (aortic catheterization), etc. Different classifications are used to describe the dissection type: De Bakey and Stanford. A total of 60% of dissections involve the ascending aorta (DeBakey I, II; Stanford A). A total of 40% are type III DeBakey, B Stanford and are divided into complicated and uncomplicated. The complicated type B AAD is associated with ruptures, organ malperfusion, and a rapid increase of aortic diameter. Type B complicated dissections need operative treatment.
G.A. Antoniu, K.El. Sakka, M. Hamady, J.H.N. Wolfe. Hybrid treatment of complex aortic arch disease with supra-aortic debranching and endovascular stent graft repair. European Journal of vascular & endovascular surgery; june 2010, volume 39 issue 6, pages 683 - 690.
Ernst Weigang, Jack Parker, Martin Czerny, Ali.A. Peyvandi, et al. Endovascular aortic arch repair after aortic arch debranching. The annals of thoracic surgery, February 2009, vol.87, issue 2, pages 603 - 607.
R.A. Yoshida, R. Kolvenbach, W.B.Yoshida, et al. Total endovascular debranching of the aortic arch. European Journal of vascular & endovascular surgery; november 2011, volume 42, issue 5, pages 627
– 630.
